Ventricular septal defect (VSD) is the most common congenital heart defect, representing abnormal communication between the right and left ventricles. While many small VSDs close spontaneously, larger defects cause significant left-to-right shunting requiring surgical or catheter-based closure. Livasa Hospital provides comprehensive VSD management from diagnostic evaluation through definitive closure with excellent outcomes.
The ventricular septum is the wall separating the right and left ventricles. VSDs are classified by location:
Membranous VSD – Most common (70% of cases); below the aortic valve
Muscular VSD – Within the muscular septum; more likely to close spontaneously
Outlet (conal) VSD – Beneath the pulmonary and aortic valves
Inlet VSD – Associated with inlet septum abnormalities
The size of the VSD determines haemodynamic significance:
Small VSDs ("restrictive"):
Minimal left-to-right shunting
Normal cardiac size and function
Low risk of complications
Often close spontaneously
Large VSDs ("non-restrictive"):
Significant left-to-right shunting
Left ventricular and atrial dilatation
Progressive pulmonary hypertension risk
Require closure before complications develop
Symptoms depend on VSD size and age:
Small VSDs:
Often asymptomatic (detected by murmur)
Normal growth and development
Large VSDs:
Poor feeding and failure to thrive
Recurrent respiratory infections
Breathlessness with exertion
Progressive fatigue
Heart failure symptoms (in severe cases)
Diagnosis and assessment includes:
Clinical examination – Characteristic murmur heard at left sternal border
Chest X-ray – Cardiomegaly and pulmonary congestion in large defects
ECG – Ventricular hypertrophy patterns
Echocardiography – Gold-standard; demonstrates defect, size, location and shunt magnitude
Cardiac catheterization – Occasionally needed for pulmonary vascular disease assessment
Conservative Management:
Small VSDs followed expectantly
Natural closure occurs in 30–50% of muscular VSDs within 2 years
Regular follow-up echocardiography
Medical Therapy:
Diuretics for heart failure symptoms
ACE inhibitors to reduce afterload
Antibiotics to prevent endocarditis
Interventional Closure:
Catheter-based closure for selected secundum-type VSDs
Minimally invasive with excellent results
Surgical Repair:
Indicated for large VSDs causing heart failure
Risk of progressive pulmonary hypertension
Failure of medical therapy
Surgical repair involves:
Median sternotomy and cardiopulmonary bypass
Right atrial or ventricular access to identify the defect
Direct closure using primary suturing or patch placement
Careful attention to nearby valves and conduction pathways
Verification of complete closure
Bypass weaning and closure
ICU monitoring for 24–48 hours
Gradual weaning from mechanical ventilation
Progressive activity escalation
Hospital discharge on postoperative day 5–7
Periodic echocardiographic surveillance
Assessment of valve function
Detection of complications
Endocarditis prophylaxis guidance
Operative mortality <1% for isolated VSD repair
Excellent long-term functional outcomes
Normal exercise capacity achievable
Life expectancy equal to general population
Experienced congenital cardiac surgeons
Specialised paediatric cardiac anaesthesia
Dedicated paediatric cardiac ICU
Access to catheter-based closure when appropriate
Comprehensive long-term follow-up
Livasa Hospital Mohali
Sector 71, Sahibzada Ajit Singh Nagar (SAS Nagar)
Mohali, Punjab – 160071
Phone: +91-80788 80788 (24/7 Emergency)
Website: www.livasahospitals.com
Call +91-80788 80788 to arrange evaluation for VSD at Livasa Hospital.
+91 80788 80788
Livasa Healthcare Group Corporate Office,Phase-8, Industrial Area, Sector 73, Sahibzada Ajit Singh Nagar, Punjab 160071
livasacare@livasahospitals.in
| Mohali | +91-99888 23456 |
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